Mutation in the Plasmodium falciparum BTB/POZ Domain of K13 Protein Confers Artemisinin Resistance

Paloque, L., Coppée, R., Stokes, B. H., Gnädig, N. F., Niaré, K., Augereau, J.-M., Fidock, D. A., Clain, J., & Benoit-Vical, F. (2022). Mutation in the Plasmodium falciparum BTB/POZ Domain of K13 Protein Confers Artemisinin Resistance. Antimicrobial Agents and Chemotherapy, 66(1). https://doi.org/10.1128/aac.01320-21

Authors:

Lucie Paloque

Jean-Michel Augereau

Francoise Benoit-Vical

Romain Coppee

Jerome Clain

Barbara H. Stokes

Nina F. Gnadig

David A. Fidock

Karamoko Niare

Affiliated Authors:

Barbara H. Stokes

Nina F. Gnadig

David A. Fidock

Columbia Affiliation:

Vagelos College of Physicians and Surgeons
- Department of Microbiology and Immunology
- Department of Medicine
  Division of Infectious Diseases

Subjects:

Malaria (OpenAlex Topic)

Computational Methods in Drug Discovery (OpenAlex Topic)

Epidemiology and Treatment of Chagas Disease (OpenAlex Topic)

Antimalarials (MeSH)

Artemisinins (MeSH)

BTB-POZ Domain (MeSH)

Protozoan Proteins (MeSH)

Author Keywords:

malaria

artemisinin resistance

k13 mutation

btb/poz domain

elink

Publication Type:

Article

Unique ID:

10.1128/AAC.01320-21

PMID:

34606334

DOI:

10.1128/AAC.01320-21

Journal:

Antimicrobial Agents and Chemotherapy

Publication Date:

2022-01-01

Data Source:

Web of Science

Source Link:

View Web of Science Record

View OpenAlex Record

Record Created:

Wednesday, February 19, 2025 - 11:45