Antibodies targeting ADAM17 reverse neurite outgrowth inhibition by myelin-associated inhibitors
Calcineurin-NFAT Signaling in Transcriptional Regulation
Subject Vocabulary
OpenAlex Topic
Unique ID
https://openalex.org/T12620
Saha, N., Chan, E., Mendoza, R. P., Romin, Y., Tipping, M. J., & Nikolov, D. B. (2025). Antibodies targeting ADAM17 reverse neurite outgrowth inhibition by myelin-associated inhibitors. Life Science Alliance, 8(6), e202403126. https://doi.org/10.26508/lsa.202403126
Columbia Affiliation:
Subjects:
Nerve injury and regeneration
(OpenAlex Topic)
Calcineurin-NFAT Signaling in Transcriptional Regulation
(OpenAlex Topic)
Adult Neurogenesis and Brain Development
(OpenAlex Topic)
ADAM17 Protein
(MeSH)
Neuronal Outgrowth
(MeSH)
Nogo Proteins
(MeSH)
Membrane Proteins
(MeSH)
Nerve Tissue Proteins
(MeSH)
Publication Type:
Article
Unique ID:
10.26508/lsa.202403126
PMID:
Journal:
Publication Date:
Data Source:
PubMed
Banerjee, D., Boboila, S., Okochi, S., Angelastro, J. M., Kadenhe-Chiweshe, A. V., Lopez, G., Califano, A., Connolly, E. P., Greene, L. A., & Yamashiro, D. J. (2023). Activating Transcription Factor 5 Promotes Neuroblastoma Metastasis by Inducing Anoikis Resistance. Cancer Research Communications, 3(12), 2518–2530. https://doi.org/10.1158/2767-9764.crc-23-0154
Columbia Affiliation:
Subjects:
Neuroblastoma
(MeSH)
Antineoplastic Agents
(MeSH)
Neuroblastoma Research and Treatment
(OpenAlex Topic)
Calcineurin-NFAT Signaling in Transcriptional Regulation
(OpenAlex Topic)
Mechanosensitive Ion Channels in Physiology and Disease
(OpenAlex Topic)
Grants:
P30CA013696
(NIH – National Cancer Institute)
R35CA197745
(NIH – National Cancer Institute)
U01CA272610
(NIH – National Cancer Institute)
S10OD021764
(NIH – Office of the Director, National Institutes of Health)
S10OD012351
(NIH – Office of the Director, National Institutes of Health)
Publication Type:
Article
Unique ID:
10.1158/2767-9764.crc-23-0154
PMID:
Journal:
Publication Date:
Data Source:
OpenAlex
Source Link:
Record Created:
Li, S., Fu, J., Liu, G., Ma, H., Mapara, M., Marcireau, C., & Lentzsch, S. (2023). P-200 MAP4K2 inhibition reinforces sensitivity to iberdomide in RAS-mutated MM through a CRBN-independent mechanism. Clinical Lymphoma Myeloma and Leukemia, 23, S145–S146. https://doi.org/10.1016/s2152-2650(23)01818-9
Subjects:
Calcineurin-NFAT Signaling in Transcriptional Regulation
(OpenAlex Topic)
Chromatin Remodeling in Cancer and Development
(OpenAlex Topic)
Targeted Protein Degradation in Biomedical Research
(OpenAlex Topic)
Publication Type:
Article
Unique ID:
10.1016/s2152-2650(23)01818-9
Publication Date:
Data Source:
OpenAlex
Source Link:
Record Created:
Abrogation of MAP4K4 protein function causes congenital anomalies in humans and zebrafish
Patterson, V., Ullah, F., Bryant, L., Li, D., Griffin, J. N., Sidhu, A., Saliganan, S., Blaile, M., Saenz, M. S., Smith, R., Ellingwood, S., Grange, D. K., Hu, X., Mireguli, M., Luo, Y., Shen, Y., Mulhern, M., Zackai, E., Ritter, A., … Bhoj, E. (2023). Abrogation of MAP4K4 protein function causes congenital anomalies in humans and zebrafish. Science Advances, 9(17). https://doi.org/10.1126/sciadv.ade0631
Subjects:
Mammalian MAP Kinase Signaling Pathways
(OpenAlex Topic)
Cell Mechanics and Extracellular Matrix Interactions
(OpenAlex Topic)
Calcineurin-NFAT Signaling in Transcriptional Regulation
(OpenAlex Topic)
Signal Transduction
(MeSH)
Zebrafish
(MeSH)
Grants:
R01HD055651
(NIH – Eunice Kennedy Shriver National Institute of Child Health & Human Development)
R01HD105868
(NIH – Eunice Kennedy Shriver National Institute of Child Health & Human Development)
K23HD088742
(NIH – Eunice Kennedy Shriver National Institute of Child Health & Human Development)
R03HD092694
(NIH – Eunice Kennedy Shriver National Institute of Child Health & Human Development)
R21TR002770
(NIH – National Center for Advancing Translational Sciences)
UL1TR002553
(NIH – National Center for Advancing Translational Sciences)
U01HG006487
(NIH – National Human Genome Research Institute)
R01AR071486
(NIH – National Institute of Arthritis and Musculoskeletal and Skin Diseases)
R01GM086537
(NIH – National Institute of General Medical Sciences)
R01MH106826
(NIH – National Institute of Mental Health)
Publication Type:
Article
Unique ID:
10.1126/sciadv.ade0631
PMID:
Journal:
Publication Date:
Data Source:
PubMed
Source Link:
Record Created:
Kucharski, M., Wirjanata, G., Nayak, S., Boentoro, J., Dziekan, J. M., Assisi, C., van der Pluijm, R. W., Miotto, O., Mok, S., Dondorp, A. M., & Bozdech, Z. (2023). Short tandem repeat polymorphism in the promoter region of cyclophilin 19B drives its transcriptional upregulation and contributes to drug resistance in the malaria parasite Plasmodium falciparum. PLOS Pathogens, 19(1), e1011118. https://doi.org/10.1371/journal.ppat.1011118
Subjects:
Antimalarials
(MeSH)
Drug Resistance
(MeSH)
Malaria, Falciparum
(MeSH)
Plasmodium falciparum
(MeSH)
Calcineurin-NFAT Signaling in Transcriptional Regulation
(OpenAlex Topic)
Epidemiology and Treatment of Chagas Disease
(OpenAlex Topic)
Global Burden of Leishmaniasis Incidence and Treatment
(OpenAlex Topic)
Publication Type:
Article
Unique ID:
10.1371/journal.ppat.1011118
PMID:
Journal:
Publication Date:
Data Source:
OpenAlex
Record Created:
Shrestha, B. R., Burgos, A., & Grueber, W. B. (2021). The Immunoglobulin Superfamily Member Basigin Is Required for Complex Dendrite Formation in Drosophila. Frontiers in Cellular Neuroscience, 15. https://doi.org/10.3389/fncel.2021.739741
Subjects:
Calcineurin-NFAT Signaling in Transcriptional Regulation
(OpenAlex Topic)
Neuroscience and Genetics of Drosophila Melanogaster
(OpenAlex Topic)
Biomedical Applications of Silk Biomaterials
(OpenAlex Topic)
Publication Type:
Article
Unique ID:
10.3389/fncel.2021.739741
PMID:
Journal:
Publication Date:
Data Source:
PubMed
Record Created:
Mechanisms of Congenital Heart Disease Caused by NAA15 Haploinsufficiency
Ward, T., Tai, W., Morton, S., Impens, F., Van Damme, P., Van Haver, D., Timmerman, E., Venturini, G., Zhang, K., Jang, M. Y., Willcox, J. A. L., Haghighi, A., Gelb, B. D., Chung, W. K., Goldmuntz, E., Porter, G. A., Lifton, R. P., Brueckner, M., Yost, H. J., … Seidman, J. G. (2021). Mechanisms of Congenital Heart Disease Caused by NAA15 Haploinsufficiency. Circulation Research, 128(8), 1156–1169. https://doi.org/10.1161/circresaha.120.316966
Columbia Affiliation:
Subjects:
Heart Defects, Congenital
(MeSH)
Role of Fibroblast Activation in Cancer Progression
(OpenAlex Topic)
Calcineurin-NFAT Signaling in Transcriptional Regulation
(OpenAlex Topic)
Molecular Mechanisms of Cardiac Development and Regeneration
(OpenAlex Topic)
Grants:
UL1TR001863
(NIH – National Center for Advancing Translational Sciences)
U01HL153009
(NIH – National Heart, Lung, and Blood Institute)
K08HL150284
(NIH – National Heart, Lung, and Blood Institute)
U01HL098153
(NIH – National Heart, Lung, and Blood Institute)
U01HL098147
(NIH – National Heart, Lung, and Blood Institute)
UM1HL098123
(NIH – National Heart, Lung, and Blood Institute)
R01HL151257
(NIH – National Heart, Lung, and Blood Institute)
UM1HL098179
(NIH – National Heart, Lung, and Blood Institute)
Publication Type:
Article
Unique ID:
10.1161/CIRCRESAHA.120.316966
PMID:
Journal:
Publication Date:
Data Source:
Web of Science